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Professor Wu Qingping

Professor Wu Qingping

Years of Experience: 35 Years

Director of the Guangdong Institute of Microbiology

Academician, researcher, and doctoral supervisor of the Chinese Academy of Engineering

Professor Wu Qingping is the recipient of a special government allowance issued by China’s State Council, and mainly engages in the research and development of biosafety monitoring and control, key technologies for the industrialization of edible fungi, and microbial fermentation engineering jobs. He is now employed as an expert of the International Food Microbiology Standardization Committee and an expert of the China Food Safety Committee.

Articles

Process For Preparing Ganoderma Spore Oil
Jan 14, 2021

Abstract

Inventor

Yizheng XieShenzhu LiBurton B. YangChong LiZhi ZhangQingping WuGuanzhou ChenBiao Luo

The present invention relates to a method for preparing Ganoderma spore oil which is in the field of biotechnology. The method includes using Ganoderma spore powder and Ganoderma powder (obtained from grinding fruiting bodies) as raw materials, applying enzymatic Ganoderma sporoderm broken methods, one-step granulation, supercritical CO2 extraction method, as well as centrifugation and refining. The light yellow oleaginous substances obtained posses a variety of physiological functions such as strengthening immunity, protecting the liver and inhibiting tumor cell growth, etc. Ganoderma spore oil prepared with the present technology contains not only spore extracts, but also extracts from Ganoderma fruiting bodies and mycelium by CO2 supercritical extraction, with more types of triterpenoids. The inhibitory effects of Ganoderma spore oil is a onefold higher than spore oil prepared from the physical preparation of the sporoderm-broken spore. Meanwhile, the problem of spore oil spoilage arising from oxidation is solved due to the low peroxide value within Ganoderma spore oil, and thereby the quality of Ganoderma spore oil is greatly improved.

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Ganoderma lucidum inhibits tumour cell proliferation and induces tumour cell death
Jan 18, 2021

Ganoderma lucidum inhibits tumour cell proliferation and induces tumour cell death

Yi-Zhen Xie a,1, Sen-Zhu Li a,1, Albert Yee c, David P. La Pierre c,d, Zhaoqun Deng c, Daniel Y. Lee c,d, Qing-Ping Wu a, Qi Chen a, Chong Li a, Zhi Zhang a, Jun Guo a, Zide Jiang b, Burton B. Yang c,d,∗

a Guangdong Institute of Microbiology, Guangdong Academy of Sciences, 100 Central Xian-Lie Road, Guangzhou, China

b Department of Plant Pathology, South China Agricultural University, 483 Wu-Shan Road, Guangzhou, China

c Sunnybrook Health Sciences Centre, 2075 Bayview Avenue, Toronto, Canada M4N 3M5

d Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Ont., Canada

Received 18 March 2005; received in revised form 8 October 2005; accepted 11 October 2005

Abstract

Ganoderma lucidum, a traditional Chinese medicinal fungus, has been a favourite remedy in oriental medicine for centuries. The objective of this study is to analyze whether G. lucidum affects cancer cell proliferation and cell death. Malignant human breast carcinoma cells were used in our studies. Different preparations of G. lucidum spores were added to the cancer cells at a final concentration of 1 mg/ml followed by incubation of the cultures for two days. Treatment with G. lucidum resulted in tumour cells detachment from the tissue culture plates and death. The proliferation of the adherent cells was also inhibited. The experiments indicated that the inhibitory effects of G. lucidum on cancer cell growth were sporoderm-broken spores (broken by enzymatic method) > sporoderm-broken spores (broken by physical method) > intact spores > buffer control....

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Ergosterol peroxide activates Foxo3-mediated cell death...
Jan 18, 2021

Ergosterol peroxide, within Ganoderma Lucidum oil, is a key bioactive compound that inhibits tumour growth by reducing reducing cancer cell survival and migration. In this study, researchers investigated how, ergosterol peroxide causes cell death among liver cancer cells.

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